|
84 |
Son SM, Song H, Byun J, Park KS, Jang HC, Park YJ, Mook-Jung I. Accumulation of autophagosomes contributes to enhanced amyloidogenic APP processing under insulin-resistant conditions. Autophagy. 2012 Aug 29;8(12).
|
Link |
|
83 |
Kook SY, Hong HS, Moon M, Ha CM, Chang SH, Mook-Jung I. Aβ1-42-RAGE interaction disrupts tight junctions of the blood brain barrier via Ca2+-calcineurin signaling. J neurosci. 2012. |
Link |
|
82 |
Kim KH, Moon M, Yu SB, Mook-Jung I. Kim JI. RNASeq analysis of frontal cortex and cerebellum from 5XFAD mice at early stage of disease pathology. Journal of Alzheimer’s disease. In press. (Co-correspondance)
|
Link |
|
81 |
Kim C, Choi H, Jung ES, Lee W, Oh S, Jeon NL, Mook-Jung I. HDAC6 inhibitor blocks amyloid beta-induced impairment of mitochondrial transport in hippocampal neurons. PLoS One. 2012;7(8):e42983. |
Link |
|
80 |
Cha MY, Han SH, Son SM, Hong HS, Choi YS, Byun J, Mook-Jung I. Mitochondria-specific accumulation of amyloid beta induces mitochondrial dysfunction leading to apoptotic cell death. PLoS One. |
Link |
|
79 |
Son SM, Song H, Byun J, Park KS, Jang HC, Park YJ, Mook-Jung I. (2012) Altered APP processing in the diabetic condition is mediated by autophagosome accumulation. Diabetes. 2012 Dec;61(12):3126-38. |
Link |
|
78 |
Lee SH, Kim KR, Ryu SY, Son SM, Hong HS, Mook-Jung I, Lee SH, Ho WK. Impaired short-term plasticity caused by mitochondrial dysfunction in dentate granule cells is the earilist synpatic deficit in a mouse model of Alzhiemer's disease. J. Neuroscience. |
Link |
|
77 |
Woo JA, Jung AR, Lakshmana MK, Bedrossian A, Lim Y, Bu JH, Park SA, Koo EH, Mook-JUng I, Kang DE. Pivotal role of the RanBP9-cofilin pathway in Ab-induced apoptosis and neurodegeneration. Cell Death & Differentiation. |
Link |
|
76 |
Jung ES, An K, Hong HS, Kim J, Mook-Jung I. Astrocyte-originated ATP protects Abeta1-42-induced impairment of synaptic plasticity. Journal of Neurosciecne. 32(9):3081-7. |
Link |
|
75 |
Moon M, Hong HS, Nam DW, Baik SH, Song H, Kook SY, Kim YS, Lee J, Mook-Jung I. Intracellular Amyloid-β Accumulation in Calcium-Binding Protein-Deficient Neurons Leads to Amyloid-β Plaque Formation in Animal Models of Alzheimer's Disease. J Alzheimers Dis. 2012 Jan 20. |
Link |
