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0 6 |
Inhibition of Glutaminyl Cyclase Ameliorates Amyloid Pathology in an Animal Model of Alzheimer's Disease via the Modulation of γ-Secretase Activity. Song H, Chang YJ, Moon M, Park SK, Tran PT, Hoang VH, Lee J, Mook-Jung I. J Alzheimers Dis. 2015;43(3):797-807. |
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0 5 |
Aβ-induced degradation of BMAL1 and CBP leads to circadian rhythm disruption in Alzheimer’s disease. Song, H., Moon, M., Choe, H. K., Han, D. H., Jang, C., Kim, A., ... & Mook-Jung I. Mol Neurodegener. 2015 Mar 19;10(1):13. |
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0 4 |
Acute ER stress regulates amyloid precursor protein processing through ubiquitin-dependent degradation. Jung, E. S., Hong, H., Kim, C., & Mook-Jung, I. Scientific reports. 2015 Mar 5;5:8805 |
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0 3 |
The role of mitochondrial DNA mutation on neurodegenerative diseases. Moon-Yong Cha, Dong Kyu Kim, and Inhee Mook-Jung. Experimental & molecular medicine 2015 Mar 13;47:e150 |
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0 2 |
β-amyloid and α-synuclein cooperate to block SNARE-dependent vesicle fusion. Choi BK, Kim JY, Cha MY, Mook-Jung I, Shin YK, Lee NK Biochemistry. 2015 Mar 10;54(9):1831-40 |
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0 1 |
Correlation between orphan nuclear receptor Nurr1 expression and amyloid deposition in 5XFAD mice, an animal model of Alzheimer's disease. Moon M, Jeong I, Kim CH, Kim J, Lee PK, Mook-Jung I, Leblanc P, Kim KS. J Neurochem. 2015 Jan;132(2):254-62. doi: 10.1111/jnc.12935. Epub 2014 Sep 16. |
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